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Previous pendulum impact tests have shown that knee joint injuries and tibial-fibular fractures may occur when loads are directed against the lower leg rather than directly against the femur in the knee. In order to further improve our understanding of lower extremity restraint mechanics, simulated frontal barrier crash experiments were conducted with unembalmed human cadavers and an anthropomorphic dummy restrained by a two-point shoulder belt. In the first test, an experimental bolster was specifically positioned so that the cadaver's lower leg would strike the bolster, thus inducing restraining loads entirely below the knee joint. The analysis of occupant kinematics showed that the flexed knee rode over and forward of the low-positioned bolster. Restraint induced considerable shearing load across the knee joint. Bolster measurements indicated a peak load of approximately 4.0 kN per leg which resulted in a contralateral central tear of the posterior cruciate ligaments.

Classical blunt thoracic impacts have involved midsternal anteroposterior loadings to an upright-positioned subject. Data on the sensitivity of human cadaver and/or animal model biomechanical and injury responses to blunt loadings at different sternal locations is needed to evaluate the efficacy of current injury-potential guidelines for nonsite-specific frontal impacts. In addition, the biomechanics and injury mechanisms associated with lateral impacts constitute a subject of increasing consideration for occupant protection. Twelve anesthetized pigs were subjected to various blunt frontal or a right-side impact to assess biomechanical and injury response differences in a living animal model.

A planar mathematical model was developed to provide means of studying factors which can influence the function of lower torso restraint via a padded lower instrument panel or knee bolster. The following factors were judged to play the most significant role: 1) initial fore-and-aft position of the seated occupant relative to the knee restraint; 2) location of the knee-to-bolster contact; 3) angular orientation of the bolster face; 4) primary axis of the bolster resisting force, 5) variations in vehicle crash parameters (e.g., toepan rotation and displacement and seat deflection); and 6) deformation characteristics of the bolster. The model of a seated occupant included radiographic and empirical data on the anatomy of the links and joints in the lower extremity.

Current finite element models of head impact involve a geometrically simplified fluid-filled shell composed of homogeneous, linear and (visco) elastic materials as the primary surrogate of the human skull and brain. The numerical procedure, which solves the mechanical response to impact, requires and presumes continuity of stress and displacement between elements, a defined boundary condition simulating the neck attachment and a known forcing function. Our critical review of the models discussed, primarily, the technical aspects of the approximations made to simulate the head and the limitations of the proposed analytical tools in predicting the response of biological tissue. The following critical features were identified as major factors which compromised the accuracy and objectivity of the models: - The brain was approximated by a fluid contained in an elastic or rigid shell with no provision for relative motion between the shell and fluid.

The following work was done in support of a continuing program to better characterize the behavior of the human chest during blunt sternal impact. Previous work on this problem has focused on determining the force-time, deflection-time, and force-deflection response of embalmed and fresh cadavers to impact by a 15 cm (6 in) diameter striker of variable mass traveling at velocities of 22.5-51 km/h (14-32 mph) and striking the sternum at the level of the fourth intercostal space. Additional questions persist concerning whether the anterior and posterior regions of the chest behave as highly damped masses or oscillate after impact, the relationship between force delivered to the surface of the body and the acceleration of the underlying regions, and the influence of air compressed in the lung on thoracic mechanics.

Life threatening chest injury can involve partial or full tears of the aorta. Investigations of fatal injuries in automobile accidents indicate that aortic trauma occurs in 10-20% of the cases. The major sites of aortic trauma include the aortic isthmus, the root, and the aortic insertion at the diaphragm - all of which are points of aortic tethering. The biomechanics of the injury process involve stretching of the vessel from points of tethering and hydrodynamic increases in blood pressure, which stretch the tissue to failure at a strain of about 150%. The non-isotropic stretch response of aortic tissue is discussed with reference to the frequent transverse orientation of the laceration. Congenital and pathophysiological conditions also influence the failure characteristics of the tissue. The significant factors associated with traumatic injury of the aorta are discussed in this review paper which is based on published technical information.

A femur fracture injury criterion is presented that assesses the dependence of the permissible human knee load on the duration of the primary force exposure. Currently a constant allowable femur load limit of 7.6 kN (1700 lb) is specified in FMVSS 208, but recently the Federal Government proposed elevating the allowable limit to 10.0 kN (2250 lb), which is in excess of the limited experimental average static femur fracture force of 8.90 kN (2000 lb). A general analysis of all of the available biomechanics data and mathematical models on femoral impact response and fracture indicates a significant load time dependence for primary pulse durations below 20 ms that can elevate the permissible femur load above the Federally proposed allowable limit of 10.0 kN (2250 lb).

Thoracic impact response and injuries of living and postmortem porcine siblings were investigated to quantify comparative differences. Thirteen male animals, averaging 61.4 kg, from five different porcine litters comprised the two animal samples. Porcine brothers were subjected to similar impact exposures for which at least one brother was tested live, anesthetized and another dead, post rigor with vascular repressurization. Statistically significant differences in biomechanical responses and injuries were observed between live and postmortem siblings. On the average the anesthetized live animals demonstrated a greater thoracic compliance, as measured by increased normalized total deflections (21% Hi), and reduced overall injuries (AIS 14% Lo and rib fractures 26% Lo) at lower peak force levels (13% Lo) than did the postmortem subjects. However, individual comparisons of “match-tested” siblings demonstrated very similar responses in some cases.

Five anesthetized porcine subjects were exposed to blunt thoracic impact using a 21 kg mass with a flat contact surface traveling at 3.0 to 12.2 m/s. The experiments were conducted to assess the appropriateness of studying in vivo mechanical and physiological response to thoracic impact in a porcine animal model. A comprehensive review of comparative anatomy between the pig and man indicates that the cardiovascular, respiratory and thoracic skeletal systems of the pig are anatomically and functionally a good parallel of similar structures in man. Thoracic anthropometry measurements document that the chest of a 50 to 60 kg pig is similar to the 50th percentile adult male human, but is narrower and deeper. Peak applied force and chest deflection are in good agreement between the animal's responses and similar impact severity data on fresh cadavers.

The concept of rate of onset as an injury potential index is critically discussed through the analysis of a wide range of noninjurious whole body decelerations and localized impacts. Examination of the physical data shows that extremely high rates of onset are tolerable without injury and that these levels of rate of onset are reciprocally dependent on the pulse rise time. The physical data is next discussed with reference to existing acceleration injury criteria, specifically the GSI and HIC indices. This work substantiates the conclusions that a single rate of onset tolerance level is not warranted and that rate of onset is not a proven injury potential index.

The introduction of energy absorbing steering systems has provided a substantial reduction of occupant injury in car crashes. However, the steering system remains the most important source of occupant injury. Injury associated with steering assembly contact is due to high exposure; energy absorbing steering systems reduce the risk of injury for drivers when compared to the injury risk of right front passengers. Our investigation addressed loading of the upper abdominal region by the steering wheel rim using a physiological model for study of soft tissue injury. Injury to the liver was related to the abdominal compression response associated with rim loading. Although liver injury correlated somewhat with peak abdominal compression, a better correlation was found when the rate of compression was also considered. Force limiting by the steering wheel, not by column compression, most strongly influenced the outcome of abdominal injury.

The NHTSA's side impact dummy (SID) was evaluated against what is known of the side impact response of the human chest and responses were compared with data on Hybrid III frontal and EURQSID side impact characteristics. The SID dummy lacks a human-like chest deflection response which is crucial to the injury indicating capability of a dummy, it has a 9.8 kg near-side rib mass which is approximately an order of magnitude greater than that of the human, and it develops impact forces that are nearly three times higher than the recommended human chest response. It possesses characteristics primarily of an inertial device. The thoracic trauma index (TTI) was evaluated as an indicator of side impact injury risk, and design trends and optimized padding characteristics identified with the SID and TTI were compared with those from the Hybrid III dummy and viscous or compression injury criteria.

Knee bolsters on the lower instrument panel have been designed to control occupant kinematics during sudden deceleration. However, a wide variability in car occupant anthropometry and choice of seating posture indicates that lower-extremity contacts with the impingement bolster could predominantly load the flexed leg through the knee (acting through the femur) or through the tibia (acting through the knee joint). Potential injuries associated with these types of primary loading may vary significantly and an understanding of potential trauma mechanisms is important for proper occupant restraint.

This study analyzed FMVSS 301 rear impact tests with an instrumented rear-seat dummy. NHTSA conducted 15 FMVSS 301 rear crash tests with an instrumented and belted 50th Hybrid III dummy in the rear seat. In series 1, there were three repeat tests with the Jeep Liberty and two others, but no onboard camera view. In series 2, there were 8 tests with 2003-2005 MY (model year) vehicles that had rear head restraints. In series 3, there were two tests with 2004-2005 MY vehicles that did not have rear head restraints. There was an onboard camera view of the rear occupant in series 2 and 3. The dummy responses were evaluated and compared to relevant IARVs (injury assessment reference values). Based on the HRMD, the average height of the rear head restraints was 80.4 ± 3.4 cm (31.6″ ± 1.3″) above the H-point. In series 1, the delta V was 24.4 ± 2.0 km/h (15.2 ± 1.3 mph).

Blunt impacts to the back of the torso can occur in vehicle crashes due to interaction with unrestrained occupants, or cargo in frontal crashes, or intrusion in rear crashes, for example. Six pendulum tests were conducted on the back of an instrumented 50th percentile male Hybrid III ATD (Anthropomorphic Test Device) to determine kinematic and biomechanical responses. The impact locations were centered with the top of a 15-cm diameter impactor at the T1 or at T6 level of the thoracic spine. The impact speed varied from 16 to 24 km/h. Two 24 km/h tests were conducted at the T1 level and showed repeatability of setup and ATD responses. The 16 and 24 km/h tests at T1 and T6 were compared. Results indicated greater head rotation, neck extension moments and neck shear forces at T1 level impacts. For example, lower neck extension was 2.6 times and 3.8 times greater at T1 versus T6 impacts at 16 and 24 km/h, respectively.

Injury and disability associated with head (brain), neck (spinal cord) and facial injury account for 61.7% of the total societal Harm in the most recent estimate of motor-vehicle related crash injuries. This paper discusses the need for accurate information on translational and rotational acceleration of the head as the first step in critiquing the Head Injury Criterion (HIC) and other injury predictive methods, and developing a fuller understanding of brain and spinal cord injury mechanisms. A measurement system has been developed using linear accelerometers to accurately determine the 3D translational and rotational acceleration of the Hybrid III dummy head. Our concept has been to use the conventional triaxial accelerometer in the dummy's head to assess translational acceleration, and three rows of in-line linear accelerometers and a least squares analysis to compute statistical best-fits for the rotational acceleration about three orthogonal axes.

The objective of this study was to analyze rear crashes for the risk of serious injury (AIS 3+) by delta V. Rear impacts were analyzed for occupants sitting in front seats of light vehicles. Data was obtained from NASS-CDS for calendar years 1991-2004. Tow-away crashes with ≤15 mph rear delta V account for 67% of rear impacts and 15% of serious injury. Even for crashes <30 mph delta V, the risk for serious injury is only 0.24% (less than 1 per 420 exposed occupants). Risks increase for higher delta Vs. Individual cases in the 1997-2004 NASS-CDS electronic database were reviewed for serious injury in crashes with ≤15 mph delta V and ≥35 mph for light vehicles with calendar year >1996 to better understand injury mechanisms. Nine cases were available where a front-seat occupant was seriously injured in ≤15 mph rear delta V impact. Most cases involved older occupants, some of whom had stenosis of the cervical spine.

A “central” theory for the biomechanics of brain injury is proposed that includes the construct that acceleration of the head, per se, is not the proximate cause of injury. Rather, rapid motion of the skull causes displacement of the hard bony structures of the head against the soft tissues of the brain, which lag in their motion due to inertia and loose coupling to the skull. Relative displacement between brain and skull produces deformation of brain tissue and stretching of bridging veins, which contribute to the tissue-level causes of brain injury. The first step in an accurate interpretation of brain injury risk in dummies involves the measurement of the three-dimensional components of translational and rotational acceleration of the head.

Role of the Seat in Rear Crash Safety addresses the historic debate over seatback stiffness, energy absorbing yielding, occupant retention and whiplash prevention; and it provides a scientific foundation for the direction GM pursued in the development and validation of future seat designs. It also describes the multi-year research study into the role of the seat in rear crash safety - first by addressing the need for occupant retention in the more severe rear crashes; and then by addressing the needs for an adequately positioned head restraint and changes in the compliance of the seatback to lower the risks of the whiplash in low-speed crashes.